rabbit anti plexinb2 (Proteintech)
93
Structured Review
Proteintech
rabbit anti plexinb2
Rabbit Anti Plexinb2, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 19 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+anti+plexinb2/Plexin+B2+Antibody/pm39401575-91-40-44
Average 93 stars, based on 19 article reviews
Rabbit Anti Plexinb2, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 19 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+anti+plexinb2/Plexin+B2+Antibody/pm39401575-91-40-44
Average 93 stars, based on 19 article reviews
rabbit anti plexinb2 - by Bioz Stars,
2026-10
93/100 stars
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other:Article Title: A balance of noncanonical Semaphorin signaling from the cerebrospinal fluid regulates apical cell dynamics during corticogenesis. Article Snippet: The following primary antibodies were used: rabbit anti-GFP (1:100; Invitrogen, A11122), goat anti-Nrp1 (1:100; R&D, AF566), goat anti-Nrp2 (1:100; R&D, AF567), rat anti-PH3 (1:50; Sigma-Aldrich, HT28), mouse anti–-tubulin (1:100; Sigma-Aldrich, GTU488), rabbit anti-PlexinA2 (1:25; Santa Cruz Biotechnology, sc-25640), rabbit anti-PlexinB1 (1:100; Abcam, ab39717), rabbit anti-PlexinB1 (1:200; antibodies-online, ABIN749228), rat anti-PlexinB2 (1:100; Invitrogen, 772417), Article Title: Microglia-astrocyte crosstalk is regulated by Astragalus polysaccharides mediated through suppression of Sema4D-PlexinB2 signaling in experimental autoimmune encephalomyelitis. Article Snippet: The crosstalk between microglia inflamed in multiple sclerosis (MIMS) and astrocytes inflamed in MS (AIMS) is a crucial factor in the formation of the central inflammatory microenvironment and neurotoxicity.. Astragalus polysaccharides (APS), an important bioactive component extracted from the dried root of Astragalus, was previously found by our team to attenuate the formation of pro-inflammatory microglia and neurological dysfunction in the experimental autoimmune encephalomyelitis (EAE) mice, a classic model of MS. To investigate the effect of APS on the MIMS-AIMS crosstalk and its underlying mechanism, in this study, a mouse model of EAE and a co-culture model of microglia-astrocytes in vitro were established.. It was discovered that APS can alleviate the neurological dysfunction of EAE mice and effectively inhibit the formation of MIMS and AIMS both in vivo and in vitro. |